L’ordonnance médicale relative aux soins à apporter à Mlle Gisèle Tientcheu disponible

Chers(es) tous/ toutes,  nous continuons à adresser nos remerciements à toutes les personnes de bonne volonté qui ne cessent de nous envoyer des courriels, de nous passer les coups de fils, de nous envoyer des fax. Parmi celles là, certaines ont pris attache directement avec la patiente au Cameroun, d’autres avec Action Solidaire International. Parmi les réactions, plusieurs ont manifesté le désir d’entrer en possession de l’ordonnance délivré par les médecins traitants du Ministère de la santé publique délivrée à Gisèle.

Nous l’avons avec nous. La loi sur la protection de la personne physique ne nous autorisant pas à diffuser le document dans son intégralité, nous avons jugés utile de le mettre à la disposition de tous ceux ou celles qui en ont besoin.

Seulement, pour entrer en possession de cette dernière, il serait nécessaire et mieux indiqué de nous adresser un courrier à l’émail de A.S.I ( iactionsolidaire@yahoo.fr) afin que nous vous faisons parvenir ledit document par voie électronique ou par voie de fax..

Les résultats du laboratoire datent du 24 juillet 2008 et comportent l’historique du traitement de la patiente.

Merci une fois de plus.

Aidez Gisèle, tout comme des milliers de personnes qui se trouvent dans sa situation au Cameroun.

Comment aider ?

Envoyez vos dons, SVP, à: Mme KEMAJOU  Charlotte Paris + 33 6 13 94 82 08

FOUDA Delphine, Colmar- France: +33 614 74 82 76

TCHANGUE Marcel , Bruxelles: + 32 475 354 970

Georges MBOTCHAK. Grande Bretagne Tel: + 44 794 492 7947

Mlle Tienctcheu Marie Gisèle, Cameroun: + 237 99 87 19 27

Ou encore...

Vos contributions peuvent également être effectuées directement sur ce compte avec pour communication : Help Gisele Tientcheu + votre nom ou initiales

Banque AXA Belgique. N° de compte :  750- 63 07 58 3- 25
IBAN :  BE 70 75 06 30 75 83 25
BIC : AXA BB E22

Communiquez nous après les références du virement ou du transfert à : iactionsolidaire@yahoo.fr

L'équipe de Action Solidaire International

Comments

[undergraduate dissertation]

Blogs are so informative where we get lots of information on any topic. Nice job keep it up!!

[TcHouangeup]

AIDS DEFINING ILLNESSES,
THEIR CAUSES AND TREATMENT

Last Update: July 2001


Treatment recommendations based on the works of Dr. Heinrich Kremer (Hamburg), Prof. Alfred Hässig (Berne), Dr. Stefan Lanka (Suttgart), Dr. Eleni Papadopulos-Eleopulos et al. (Perth), Dr. Leonore Herzenberg et al. (Stanford University), and Furchgott and Ignarro (avaiable at www.ncbi.nlm.nih.gov)

The many and varied diseases that can define the AIDS syndrome: fungal infections of the lung, of the mucous membranes, the brain, and the gut, and the degenerative changes in the endothelial cells of blood vessels and lymphatic vessels (Kaposi Sarcoma), occur because of an ongoing heightened production of gaseous nitricoxide and oxygen radicals in immune cells and other cells. Under these conditions CD4 helper cells mature predominantly to cells with TH2 cytokine prophile, which migrate to the bone marrow, where they activate defences against bacteria by producing antibodies, but only few mature to TH1 cells mesurable in plasma, which activate the dedection and destruction of fungus and virus infected cells and of altered cells. If this situation persists, a higher quantity of proteins of the cytoskeleton and of mitochondria is released as en effect of heightened cell decay. Against these proteins a higher rate of the antibodies are formed. This antibodies and antibodies that occure in hepatitis and due to toxic pollution are detected by the HIV-antibody tests. Once a certain, arbitrary level is reached, the patient is declared "HIV positive".

A elevated level of nitricoxide and oxygen radicals comes about as a result of:

- ongoing contact with antigens (e.g. from repeated or chronic infections, injuries, operations and dirty water).

- repeated contact of foreign proteins to the plasma (from coagulation proteins in blood preparations and from semen liquid in unprotected anal intercourse)

- contact to toxic substances in food (e.g. aphlatoxines in wet cereals), medicaments and from environment pollution, toxic decomposition products from modern chemicals (heavy metals (e.g. carrier substances in hepatitis B-vaccines, amalgan fillings)

- inhalation of nitrites ("poppers") which are stored in cells as NO2. They are released through physical exertion on increased exposure to calcium ions. This affects the endothelial cells of blood vessels and lymphatic vessels with a small capillary diameter, and leads thereby to degenerative changes (swollen lymph nodes and finally to Kaposi Sarcoma).

- impairment of the mitochondria, the single cell energy suppliers, which synthesize the energy-carrying-molecule ATP, used for all functions of the organism

The causes of chronic mitochondrial damage are:

- damage of the mitochondrial DNA due to antibiotics (e.g. sulpha compounds such as cotrimoxazol, TMPSMX) which block the synthetisation of folic acid and purine, and lead thereby to the exhaustion of the mitochondrial thiol-pool. Likely effects are caused by heavy metals and by cytostatics like AZT. All this substances bind the SH-groups of glutathione and cysteine and impair thereby the activity of mitochondria.

- reduced glutathione produced resulting from liver damage, e.g. chronic hepatitis (occuring frequently in gay men, hemophiliacs and intravenuous drug consumers), excessive alcohol consumption, or through shortage of nutritional cysteine, esp. in developing countries. Glutathion molecules reduce oxygen- and nitricoxyde-molecules, so that ATP production in mitochondria is not disturbed. An ongoing shortage of glutathione means that phagozyte poison themselves attacking fungi and virus containing cells by means of NO.

- reduced oxygen transport in cells because of oxidation (methhaemoglobinaemia) which exceeds the reductive capacity of glutathione. This comes about because of the strongly oxidising effect of nitrites (poppers), antibiotics (cotrimoxazol, TMPSMX) and insecticides (e.g. Lindan in moistures against crab louse), nucleoside analogues (e.g. AZT), heavy metals and chemicals.

- lack of plant antioxidants which bind to toxic degradation products (oxygen radicals) and thereby reduce inflammation and stressreactions.

On prolonged impairment of mitochondria, they dissolve their symbiosis with the host ("Warburg Phenomenon"). Cells then increasingly switch over to producing energy by anaerobic fermentation, which results in excess lactic acid production, and the growth of fungi and opportunists, and ultimately to wasting, at which point cells obtain essential nutrients directly from the myoproteine. By an heightened activity of reverse transcription the cellnucleus then saves its genotype. Continuous activation of macrophages leads in this situation to an ongoing release of messanger substances (Inteleukine 2) which trigger the release of stress-hormones in the adrenal gland. This hormones induce the formation of TH2 CD-4 cells, that activate the formation of antibodies in the bone marrow, whereas cellular immune reactions induced by TH1 cells are continuously suppressed.

By means of:

- A supply of sulphur compounds in sea salt, mineral water and algal products, and of cysteine and methionine containing protein mixtures, (Cysteine, N-acetyl-cysteine and arginin, (3-8 gramme daily)also in curd and whey) and folic acid (300 miligramme daily) can stimulate glutathione formation in the liver. Glutathione must be administered in the mean time intravenously (600 milligramme daily) untill its formation in the liver works sufficiently again.

- Plant antioxidants, e.g. PADMA 28 (2-3 times 2 tabletts daily) or artemisia annua in UWEMBA pastilles (available from www.nusag.com) which bind to toxic oxygen decay products, and natural protease inhibitors (heparine and heparinoids in agar, algae or cartilage preparations), which activate the body's own anti-proteases and bind to cations that attack the cell walls, slow down chronic inflammatory reactions going allong with increased cell division.

- Co-enzyme Q10 and NADH and high doses of Vitamin C and E can improve electron transport in the respiratory chain of cells. Folic acid (300 milligramme daily), thiols L-carnitin and low doses of selenium, (e.g. brewers'yeast), and zinc can support the synthetisation of ATP in mitochondria and the repair of damage to mitochondrial DNA.

- Opportunistic infections (fungy, PCP and others can be treated by omega-3 fatty acids in fishoil (3 tablespoons daily) In dificult cases gamma globulin, selective cyclo-oxygenese-2 inhibitors and difluoromethylornithine as a polyamine inhibitor can be administrated. The activity of killer cells and neutrophillia can be supported by the administration of glutamine (40 grammes daily) and L-Arginin (20-30 grammes daily).

- DHEAS (200 milligrammes daily) can diminish ongoing stress reactions in the immune system (TH1-TH2-switch) caused by the release of stresshormones (cortisol) in the adrenal gland.

- Essential fatty acids in linseed oil, thistle oil, soya oil and omega-3 fatty acid in fish oil mixed with curd, which enhighten the uptake of oxygen in cells

- Carduus marianus or Aloe vera to support the liver and partly fermented beverages that can restore the gut flora

- Ethereal oils, rubbed on to the chest and in the armpits serve to stimulate the immune system through the ground substance (matrix)

- Extract of grape fruit kernels, gargling with honey/vinegar and sulphur containing moistures or Melaleucae Alternifolia oil as a local treatment against fungal infection.

- Targeted stress reduction techniques, e.g. autogenic training, stretching and massages, and refraining from excesive physical exercise (using perfomance-enhancing drugs, e.g. coffee, alcohol, nicotine, amphetamines (X-tasy), cocaine, heroin and poppers.)

- avoiding inflammatory reactions and infections by avoiding injuries and the contact of foreing proteins to the plasma (e.g. by protection in anal intercourse).

- of a nourishment poor in sugar amd acid but rich in roughage and bases, with much high-value carbohydrates and potatoe, plant antioxidants, e.g. vegetables, fruit, herbal and green teas, cold-pressed oils, partly fermented dairyproducts, algae, soya beans, and fish but not iron-rich red meat.

...a flexible resistance in people with AIDS defining illnesses can be restored.

If limited administration of antibiotics is necessary, this basic therapy has to be continued. The treatment can be adapted to the individueal illnesses occuring. Progress achieved by these measures to bolster the immune system can be monitored by measuring stress hormone profiles, the T4/T8 cell ratio, macrophage activation (neopterine test) and cutaneous anergy, the glutathione level in plasma and in T-4 helper cells.

HIV, which is held to be responsible for causing 30 different AIDS-defining diseases, has never been shown to be transmissible nor self-reproducing; it has never been isolated, photographed or otherwise properly characterised, as required by the established rules of virology. The original experimental technique of Gallo and Montagnier in 1984 on which the HIV-antibody-tests were constructed, involved culturing cells from AIDS patients with leucaemic cells and embryonal cells, that show a high activity of reverse transcription. This effect of an artificially amplified reverse transcription was then interpreted as signifying the presence a new virus. A virus-specific enzyme could not be aprooved according to the established rules. Synthetic protease inhibitors, which are supposed to inhibit the formation of essential viral building blocks, over time, cause malaise, diabetes, kidney stones and liver failure in patients given them. After PIs and nucleoside analogues are first given, an apparent decline in inflammatroy reactions and „virus production“ may be observed, but it then rises again, which is attributed to resistance developping. Nucleoside analog drugs, that destroy for a limited time through cytostatic effects bacteries and fungy, are only ever 1% incorporated into the cell nucleus, where they should work as DNA terminators against HIV. As it has been demonstrated by animal trials since 1990 they cause irreversible damage to the mitochondrial DNA and thereby damage to the brain, the bone marrow, the muscles and internal organs and a lasting decrease of CD-4 and CD-8 cells causing opportunistic infections (cytomegalie, herpes simplex, PCP and hepatitis non-A-non-B) that define the AIDS-syndrome.

Study Group for AIDS therapy
c/o Felix A. de Fries
Eglistr. 7 CH-8004 Zürich
Tel./Fax: 0041 1 401 34 24
E-mail: felix.defries@bluewin.ch